Postinflammatory hyperpigmentation
Postinflammatory hyperpigmentation د پوستکي یو حالت دی چې د سوزش وروسته د رنګ زیاتوالي له امله څرګندېږي. Postinflammatory hyperpigmentation کیدای شي د لمر د اوږد تماس، سوزش، یا د پوستکي نورو ټپونو له امله رامنځته شي، په شمول د ځوانۍ پورې اړوند. هغه خلک چې تور پوستکي لري عموماً د لمر د ډېر تماس سره د هایپرپیګمینټیشن خطر زیات لري.
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ReferencesPostinflammatory hyperpigmentation (PIH) د پوستکي مکرره ستونزه ده چې د پوټکي سوزش یا ټپي کیدو وروسته پیښیږي. دا د اوږدې مودې لپاره دوام کوي او په هغو خلکو کې ډېر خراب دی چې د پوستکي تور رنګ لري (Fitzpatrick skin types III–VI). که څه هم دا اکثره په خپله ښه کېږي، خو وخت نیسي؛ نو درملنه اکثره د اوږدې مودې لپاره اړینه وي. د مختلفو درملنو ترکیب غوره کار کوي.
Postinflammatory hyperpigmentation (PIH) is a common acquired cutaneous disorder occurring after skin inflammation or injury. It is chronic and is more common and severe in darker-skinned individuals (Fitzpatrick skin types III–VI). While the condition typically improves spontaneously, this process can take months to years, necessitating prolonged treatment. Combination therapy is the most effective.
Postinflammatory hyperpigmentation (PIH) is a common acquired cutaneous disorder occurring after skin inflammation or injury. It is chronic and is more common and severe in darker-skinned individuals (Fitzpatrick skin types III–VI). While the condition typically improves spontaneously, this process can take months to years, necessitating prolonged treatment. Combination therapy is the most effective.
Postinflammatory hyperpigmentation د پوستکي التهاب یوه عامه پایله ده. دا د تیاره پوستکي خلکو ته ډېر سخت او په مکرر ډول اغېز کوي. مطالعې ښيي چې د postinflammatory hyperpigmentation په څېر مسلې د اصلي لاملونو څخه دي چې ولې د پوستکي تور رنګ لرونکي خلک د درملنې غوښتنه کوي. لومړنۍ درملنه د postinflammatory hyperpigmentation د حل لپاره خورا مهمه ده او عموماً د لومړني التهاب د کنټرول سره پیل کېږي. د درملنې لومړۍ کرښه عموماً د سرې اجنټونو کارول شاملوي چې د لمر سکرین سره د پوستکي رنګ روښانه کوي. دا اجنټان، لکه hydroquinone, azelaic acid, kojic acid, arbutin, licorice extracts، کولی شي په مؤثره توګه رنګ کم کړي. برسیره پردې، retinoids, mequinol, ascorbic acid, niacinamide, N‑acetyl glucosamine، او soy د ډی‑پیګمینټ ایجنټونو په توګه هم کارول کېږي. که څه هم سطحي درملنه عموماً د سطحې کچې هایپرپیګمینټیشن لپاره مؤثره ده، پروسیجرونه (laser, chemical peel) ممکن د سختو قضیو لپاره اړین وي. دا مهمه ده چې د دې درملنې په وخت کې احتیاط وکړئ ترڅو د postinflammatory hyperpigmentation د خرابېدو او رنګ زیاتېدو مخه ونیسئ.
Postinflammatory hyperpigmentation is a common sequelae of inflammatory dermatoses that tends to affect darker skinned patients with greater frequency and severity. Epidemiological studies show that dyschromias, including postinflammatory hyperpigmentation, are among the most common reasons darker racial/ethnic groups seek the care of a dermatologist. The treatment of postinflammatory hyperpigmentation should be started early to help hasten its resolution and begins with management of the initial inflammatory condition. First-line therapy typically consists of topical depigmenting agents in addition to photoprotection including a sunscreen. Topical tyrosinase inhibitors, such as hydroquinone, azelaic acid, kojic acid, arbutin, and certain licorice extracts, can effectively lighten areas of hypermelanosis. Other depigmenting agents include retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, and soy with a number of emerging therapies on the horizon. Topical therapy is typically effective for epidermal postinflammatory hyperpigmentation; however, certain procedures, such as chemical peeling and laser therapy, may help treat recalcitrant hyperpigmentation. It is also important to use caution with all of the above treatments to prevent irritation and worsening of postinflammatory hyperpigmentation.
Postinflammatory hyperpigmentation is a common sequelae of inflammatory dermatoses that tends to affect darker skinned patients with greater frequency and severity. Epidemiological studies show that dyschromias, including postinflammatory hyperpigmentation, are among the most common reasons darker racial/ethnic groups seek the care of a dermatologist. The treatment of postinflammatory hyperpigmentation should be started early to help hasten its resolution and begins with management of the initial inflammatory condition. First-line therapy typically consists of topical depigmenting agents in addition to photoprotection including a sunscreen. Topical tyrosinase inhibitors, such as hydroquinone, azelaic acid, kojic acid, arbutin, and certain licorice extracts, can effectively lighten areas of hypermelanosis. Other depigmenting agents include retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, and soy with a number of emerging therapies on the horizon. Topical therapy is typically effective for epidermal postinflammatory hyperpigmentation; however, certain procedures, such as chemical peeling and laser therapy, may help treat recalcitrant hyperpigmentation. It is also important to use caution with all of the above treatments to prevent irritation and worsening of postinflammatory hyperpigmentation.
